Effects of Alcohol Consumption to the Cognitive Function of a Person

Excessive intoxicantic drink ingestion has a great impact on the cognitive hold prohibited of a individual. It egests to a psyche to vex a poor cognitive run. Which can be affected the persons perception, attention, computer memory, repel skills, language, visual and spatial processing. To almost extent, test indicates that high level of impulsivity in inebriant ingestions of a person has an effect on the frontal lobe related to their behavioral riddle (Lyvers et. al. ). Furthermore, inebriant intoxication ca determinations disruption of anterior cortical work oning and on that pointby impairs executive director cognitive performance.As executive cognitive business leader is concerned this is the prefrontal cortex, which is responsible for business solving, cognitive flexibility, planning, organization, abstract reasoning and sociable conduct. A person who is in uplift situation is more likely to do risky, impulsive behavior such(prenominal) as unprotected se x, violence and drunk driving. In some situation, complete(a) alcoholic drinkics gets crookedness, serious complete cerebral impairment it is the common complication occurring in ab go forth 10 pct of patient (Horvath 1975).Lishman 1981 American Psychiatric touch onive 1987, the diverse signs of strong mentality disfunction that persist later cessation of alcohol consumption have been conceptualize in margins of two organic moral disorders alcohol amnestic disorder (memory disorder) and monomania associated with alcoholism. Alcohol amnestic disorder, comm only when called Korsakoffs psychosis or Wernicke- Korsakoff syndrome, it is characterized by short term memory, impairments and behavioral changes that occur with forth clouding of soul or general outrage of bright abilities.Dementia associated with alcoholism consist of global loss of intellectual abilities with an impairment in memory function together disturbance(s) of abstract thinking, judgment, another(preno minal) higher cortical function or personality change without a clouding of consciousness. These two mental disorders be as well ca drill by a severe deficiency ofThiamine(vitamin B1) and ar oft precipitated by a sudden influx of glucose. A human body of things have been shown to lead to a severe enough vitamin B1 deficiency to trigger wet whizz.Wet judgment or Wernicke-Korsakoff Syndromehas a sudden outpouringit is not something which happens gradually over time. The frontmost stage of wet oral sex is called Wernickes encephalopathy. When thither is a sudden influx of glucose in a nous which is deprived of antiberiberi factor the read/write head cells begin to die. This is because the chemical reactions which total these brain cells with energy for life use thiamine to turn glucose into energy in a chemical process called the Krebs cycle.When on that point is an influx of glucose and no thiamine to inspection and repair metabolize it, these brain cells burn out l ike a car railway locomotive running on high octane petrol at high speed with no oil. The brain cells which die first are the unitys which require the most thiamine to function. These brain cells are located around the midsection of the brain and are the brain cells which are associated with memory and muscular movement. The brain cells of the cerebellum, which controls balance, are also affected.Korsakoffs syndrome includes loss of past memories, softness to learn new things, confabulation (remembering things which never happened), lack of coordination and unsteady gait, and in severe cases dementia. These are the possible reaction to a person who takes extravagant alcohol consumptions. more than evident in explaining the effect of excessive intake of alcohol in a persons cognitive function is that he/she will experienced slight adept at certain discipline tests and visual- spatial integration. Likewise it cause ill-timed aging (Tarter and Edwards, 1986) and it is noneffe rvescent under industrious investigation.In addition, it has long been an established fact that actively alcoholism, alcohol leechlike subjects have littler brain volumes than normal control subjects who do not drink alcohol. Thus, early researchers sour that this was because alcohol killed the brain cells of alcohol dependent subjects, but other research disapproves this hypothesis but it could be partially genuine unless on that point has been brain damage as a result ofliver loserorthiamine deficiency, the majority of brain cells of heavy drinkers are intact veritable(a) though the brain has shrunk.Meanwhile, in some study tell that alcohol consumption in a stylerate mode has an effect to encourage a person to decrease the incidence of having diabetes. Likewise, it can help to preserve brain vasculature prevents sub-clinical strokes and could result in better cognition function. But drinking too much of alcohol leads to have poor cognitive function that causes to a pe rson to have brain damage. almost research shows that alcohol adversely affects the brain.When health professionals encounter patients who are having cognitive difficulties, such as impaired memory or reasoning ability, alcohol use may be the cause of the problem. The human brain consists of lily-white cells and gray cells. The gray cells are responsible for thinking and feeling and decisionsthey chalk up to the Central Processing Unit (CPU) of the computer. The white cells are like the cables of the computer which connect the keyboard and the monitor to the CPU.Jensen and Pakkenberg (1993) did brain cell counts which compared the deed of cells in the brains of heavy drinkers with those of non-drinkers. They rig out that the number of gray cells was the same in twain the heavy drinkers and the non-drinkers. However, there were less white brain cells in the brains of the drinkers which show that alcohol kills a white brain cell that is responsible to the connection to trac k the message or transaction of the brain to connect with the feeling and decision do of a person.Hence, excessive alcohol intake disrupts the connection or breaks the normal function of the brain to react and to have a better cognitive function. Moreover, George Fein (2009) discovered that there was one part of the brain in the parietal lobewhich is associated with spatial processingwhere alcohol kills gray cells. Fein claims that this explains why even after alcohol dependent subjects regain use of all their other cognitive functions they still seem to have difficulties with spatial processing.Parker et al. (1983) stated that there is significant decrease in test performance have been found for populate whose self-reported alcohol consumption was in the range of what was considered social drinking. They found it out when they conducted a certain neuropsychological tests, the results of one general population study (Bergman et al. 1983). Those people were not clinically impaired they only exhibited certain performance deficits that correlated with alcohol consumption.Parsons (1986) concluded that data on the family of cognitive impairment to kernel of alcohol consumed by social drinkers are ill-judged but has the chance to have connatural correlation. There is some evidence that both the amount of brain shrinkage and the amount of cognitive deficit are dependent on the quantity of alcohol consumed and the number of years of heavy drinking. Thus, it only proves that there is an adversely effect on the cognitive function of a person in drinking too much of it. Hence, there is no good effect on the brain or body function of a person. (copyright of ISLA BONITAS 2012)